Idan segev biography of rorys baby
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Rabbi Robert N. Levine, DD, led Congregation Rodeph Sholom for three decades, its tenth Senior Rabbi from 1991 to 2021. An inspiring teacher, speaker, counselor, and frequent guest of local and national media, he has been beloved by congregants and community alike.
Rabbi Levine was ordained at the Hebrew Union College-Jewish Institute of Religion in 1977 and received his Doctor of Divinity Degree in March 2002. He is a Phi Beta Kappa graduate of Columbia College.
Rabbi Levine is the author of three books. What God Can Do For You Now: For Seekers Who Want to Believe; Where Are You When I Need You? Befriending God When Life Hurts; and There Is No Messiah and You’re It: The Stunning Transformation of Judaism’s Most Provocative Idea.
Especially active in communal affairs, he is a past President of the New York Board of Rabbis, as well as having served as Vice-President and Chairman of its Interfaith Committee. He was Chair of the Catholic-Jewish Dialogue with the Archdiocese of New York, the publications committee of the Central Conference of American Rabbis, and is a member of the Commission of Religious Leaders of New York City, the American Jewish Committee, and Synergy/UJA Federation. He served on the Planned Parenthood Federation of America Clergy Advocacy Board
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Summary
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Young Human Cholinergic Neurons Respond to Physiological Regulators and Improve Cognitive Symptoms in an Animal Model of Alzheimer’s Disease
Introduction
The basal forebrain (BF) region is a broad topographic term describing a heterogeneous set of cellular structures on the medial and ventral cerebral hemisphere. It comprises the nucleus basalis of Meynert (NBM), the vertical and horizontal diagonal bands of Broca and the medial septal nucleus (Mesulam, 2013). This complex region contains magnocellular neurons that provide the major cholinergic projections to the cerebral cortex, hippocampus and amygdala. The BF cholinergic system has been implicated in the modulation of an ever-expanding set of behavioral states, including attention, memory and learning (Ferreira-Vieira et al., 2016). Importantly, the degeneration and loss of cholinergic neurons within the BF, especially in the NBM, represents a pathological correlate of the well-documented cholinergic derangement in Alzheimer’s disease (AD) patients (Kilimann et al., 2014).
Despite its implications as pathological substrate for cholinergic deficiency in AD brain, very little is known about the developmental properties of the NBM neuronal contingent during embryogenesis. As we move towards an era of stem cell-based